Often times infertility issues stem from or involve thyroid-endocrine pathways.

• Reproductive hormones and cortisol have the potential
• to alter the pituitary-thyroid axis
• to alter the percentage of thyroid-binding proteins
• impact thyroid hormone synthesis
• down-regulate peripheral hormone conversion
• promote thyroid receptor resistance
• Doing the right lab work is the only way to distinguish between the patterns so that the specific mechanism involved can be treated.